Allergic Diseases: Diagnosis and Treatment (Current Clinical by Phil Lieberman, John A. Anderson

By Phil Lieberman, John A. Anderson

During this up-to-date 3rd version, specialist physicians gather undemanding, clinically-oriented courses to help within the day by day administration of the allergic sufferer. With an emphasis on universal allergic illnesses and the pharmacological instruments to regulate them, this ebook disseminates the sensible wisdom that practitioners have amassed via perform and educating. 3 new chapters specialize in antihistamines, antileukotrienes, and youngsters experiencing recurrent respiration tract infections. This ebook is meant for the front-line healthcare professional who cares for allergic sufferers. it's going to aid any health care professional increase the care provided to these sufferers.

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Plant Pollens • Pollens responsible for seasonal allergies are light and transported by wind. They emanate from plants with small, nondescrite flowers. • The “pollen season” varies by type of plant and geographic location but generally follows the order of trees, then grasses, then weeds. • Airborne concentrations of pollen vary by weather conditions, with warm, dry, breezy conditions being favored for pollen dispersal. 42 Sicherer and Eggleston Fig. 1. Morphological features of several common types of pollen grains.

The neck must be palpated to search for adenopathy. At the same time, the thyroid gland should be assessed, because thyroid hormone imbalance can confound allergic symptoms. In patients with wheezing, the larynx should be auscultated to rule out stridor as an upper airway origin. Accessory muscle use of the sternocleidomastoid muscles should not be missed because it is another sign of marked airway obstruction. Lung examination is particularly relevant in the asthmatic. Configuration of the chest wall should be noted; in particular, pectus excavatum, kyphosis, lordosis, and scoliosis should be ruled out by inspection.

After another wash to remove the unbound, labeled anti-IgE, the quantity of labeled IgE on the solid phase is measured and converted into units of IgE by comparison to a standard curve. A variety of commercial assays is available. Most are accurate to a concentration of less than 5 IU/mL (12 ng/mL) of IgE and reproducible within 10% or better. Serum concentrations of IgE vary widely in normal individuals (Table 3). IgE levels are very low at birth and gradually increase, peaking in the second decade of life, followed by a slow decline into old age.

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