Acute Coronary Syndromes, Third Edition by Eric Topol

By Eric Topol

With distinctive contributions from forty five popular foreign gurus, this 3rd version stands by myself because the such a lot finished, modern view of the biology, body structure, and administration of acute coronary syndromes (ACS)-offering 32 chapters that span the state-of-the technology within the box and supply exciting discussions on CRP and different ailment markers, microcirculation, stem phone treatment, and the advance of specialised facilities for ACS therapy.

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Extra resources for Acute Coronary Syndromes, Third Edition

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Downregulation by tumor necrosis factor-alpha of monocyte CCR2 expression and lnonocyte chemotactic protein- 1-induced transendothelial migration is antagonized by oxidized low-density lipoprotein: a potential mechanism of monocyte retention i n atherosclerotic lesions. 5: I 15- 123. 83. Barks JL, McQuillan JJ, lademarco MF. TNF-alphaand IL-4 synergistically 84. 85. 86. 87. 88. 89. 90. 91. 92. 93. 94. 95. 96. increasevascularcelladhesionmoleculeI expression in cultured vascular smooth muscle cells.

Bacurcle PA, W d i AK. Neumeier D. Dysregulation of monocytic nuclex factor-kappa B by oxidized low-density lipoprotein. Arterioscler Thromb Vasc B i d 1997;17:1901-1909. Kranzhofer R, Browatzki M, Schmidt J, Kubler W. Angiotensin I1 activates the proinflammatorytranscription factor nuclear factor-kappaB in human nlonocytes. Biochem Biophys Res Comnun 1999;257:826-828. Yerneni KK, Bai W, Khan BV, Medford RM. NatarajanR. Hyperglycemiainduced activation of nuclear transcription factor kappaB i n vascular smooth muscle cells.

Nuclear factor K-B (NF-KB), Peroxisome proliferator-activated receptor activators (PPARs), CD40 receptor and its ligand, and inducible cyclooxygenase enzyme (Cox-2) are avidly being investigated as we attempt to discover the final common pathway of inflammation and its role in atherosclerosis. NF-KB NF-KB is a transcription factor located in the cytoplasm of many cells as an inactivecomplexassociated with a specific class of inhibitoryproteins, called IKB. This complexbinds and prevents nuclear translocation and DNA binding of NF-KB (102).

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